This article shows current findings related to cerebrovascular reactivity(CVR) as a marker for dementia onset and progression. We've been promoting photobiomodulation with both the Cognitolite, Vielight and other LLLT devices as a means of decreasing vascular reactivity which in our case we've referred to as 'vascular inflexibility'. Not unlike how the healthy flexibility of the cardiac rhythm is undermined by various factors that lead to infarcts and other problems, we see cerebral vasculature as being susceptible to similar challenges. Neurofeedback training will help sustain the improved level of functioning by optimizing cortical electrical functions that will help improve vascular responsiveness.

https://www.psychiatryadvisor.com/home/topics/neurocognitive-disorders/alzheimers-disease-and-dementia/cerebrovascular-reactivity-predicts-cognitive-impairment-independent-of-ad/?utm_source=newsletter&utm_medium=email&utm_campaign=pa-update-hay-20200824&cpn=&hmSubId=6Qh5EgxRQ0A1&hmEmail=VFLBRZ7KAOgTOytGd7O2WyfFDtHnYy33zUsxQGbtptc1&NID=&email_hash=24523e9d8526aea879f1cbe868535f18&mpweb=1323-102963-6650987

I tested positive for cv19 in early April and was tested as negative on 4/27 and donated plasma the next day. I was sick for 2+ weeks and managed to avoid hospital and lasting damage to my lungs and brain by adding two things to my treatment regimen, high dose Vitamin C and near infrared stimulation to my brain. I took 5 doses of clarithromycin and some promothiazine with codeine over 3 days. I spiked 103 several times and otherwise ran between 99-102 for 2 weeks. I'm quite sure the Vit C which I took about 6-10g/day saved me a lot of damage. 

Here's a further discussion of the value of Vitamin C from Medium -
​ https://link.medium.com/RmfCaNNG18

The new article in Wired on Network Neuroscience's contributions to our deeper understanding of how the brain really works is a very useful introduction to this facinating new field that helps integrate biomedical engineering with cognitive neuroscience. It is important to note that the tools they are using are mostly functional measures, of water and blood flow to identify network activity. What I hope they will begin to use quantitative EEG dynamics more and more to elucidate the mechanisms of action in brain networks at rest and under task.  Our research in this area has provided a model for understanding individual response patterns in terms of variations in dominant frequency activity. When measuring the EEG at rest we can start to see specific patterns of electrical activity that results in a particular frequency being produced. The actual frequencies are constrained both in terms of absolute range but also their variability within that range. For example, person with generalized anxiety disorder may generate a pattern of activity that is limited to between 6-12 hz with occasional bursts of higher frequency activity in the mid to upper 20hz range, while the person with major depression generates more 2-7hz activity with very little variability. We can measure this activity without using radioactive tracers and extremely expensive devices that suck up tremendous amounts of energy and some often requiring large amounts of Helium.

We look forward collaboration with network neuroscientists and providing them with the other side of the neuronal equation where you work to understand and then influence the brain's electrical activity in order to modify its chemistry.

click here for the Wired article: ​https://www.wired.com/story/a-radical-new-model-of-the-brain-illuminates-its-wiring/